Нейростоматология. Синдромы
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Trigeminal Neuralgia [14] 7 Surgery Surgery may be recommended, either to relieve the pressure on the nerve or to selectively damage it in such a way as to disrupt pain signals from getting through to the brain. In trained hands, surgery has been reported to have an initial success rate approaching 90 percent. However, some patients require follow-up procedures if a recurrence of the pain begins. Of the five surgical options, the microvascular decompression, also known as the Janetta procedure [15] , is the only one aimed at fixing the presumed cause of the pain. In this procedure, the surgeon enters the skull through a 25-millimetre (1 in) hole behind the ear. The nerve is then explored for an offending blood vessel, and when one is found, the vessel and nerve are separated or "decompressed" with a small pad, usually made from an inert surgical material such as Teflon. When successful, MVD procedures can give permanent pain relief with little to no facial numbness. Three other procedures use needles or catheters that enter through the face into the opening where the nerve first splits into its three divisions.Excellent success rates using a cost effective percutaneous surgical procedure known as balloon compression have been reported [16] This technique has been helpful in treating the elderly for whom surgery may not be an option due to coexisting health conditions. Balloon compression is also the best choice for patients who have ophthalmic nerve pain or have experienced recurrent pain after microvascular decompression. Similar success rates have been reported with glycerol injections and radiofrequency rhizotomies. Glycerol injections involve injecting an alcohol-like substance into the cavern that bathes the nerve near its junction. This liquid is corrosive to the nerve fibers and can mildly injure the nerve enough to hinder the errant pain signals. In a radiofrequency rhizotomy, the surgeon uses an electrode to heat the selected division or divisions of the nerve. Done well, this procedure can target the exact regions of the errant pain triggers and disable them with minimal numbness. Stereotactic radiation therapy The nerve can also be damaged to prevent pain signal transmission using Gamma Knife or a linear accelerator-based radiation therapy (e.g. Trilogy, Novalis, CyberKnife). No incisions are involved in this procedure. It uses very precisely targeted radiation to bombard the nerve root, this time targeting the selective damage at the same point where vessel compressions are often found. This option is used especially for those people who are medically unfit for a long general anaesthetic, or who are taking medications for prevention of blood clotting (e.g., warfarin,heparin, aspirin). A prospective Phase I trial performed at Marseille, France, showed that 83% of patients were pain-free at 12 months, with 58% pain-free and off all medications. Side effects were mild, with 6% experiencing mild tingling and 4% experiencing mild numbness. [17] There has only been one prospective clinical trial for surgical therapy for trigeminal neuralgia. In a prospective cohort trial, microvacular decompression was found to be significantly superior to stereotactic radiosurgery in achieving and maintaining a pain-free status in patients with trigeminal neuralgia and provided similar early and superior longer- term patient satisfaction rates compared with those treated with stereotactic radiosurgery [18] Social consequences of trigeminal neuralgia Most suffers of TN do not present with any outwardly noticeable symptoms, though some will exhibit brief facial spasms during an attack. Some physicians will seek a psychological root cause rather than a physiological abnormality. This is especially true of those suffering from atypical TN, who may not have any compression of the TN and in whom the sole criterion of the diagnosis may be the complaint of severe pain 8 (constant electric-like shocks, constant crushing or pressure sensations, or a constant severe ache) and in this case trigeminal neuralgia still exists but is not visible to physicians because it was caused by the nerve being damaged during a dental procedure such as root canals, extractions, gum surgeries or it may be a condition secondary to multiple sclerosis. Many TN sufferers are confined to their homes or are unable to work because of the frequency of their attacks. It is important for friends and family to educate themselves on the intense severity of TN pain and to be understanding of limitations that TN places upon the sufferer.However, at the same time, the TN patient must be extremely proactive in furthering his or her rehabilitative efforts. Enrolling in a chronic pain support group, or seeking one-on-one counseling, can help to teach a TN patient how to adapt to the newfound affliction. As with any chronic pain syndrome, TN not being the exception, clinical depression has the potential to set in, especially in younger patients who often are undertreated for chronic pain. Friends and family, as well as clinicians, must be alert to the signs of a rapid change in behavior and should take appropriate measures when necessary. It must be constantly reinforced to the sufferer of TN that treatment options do exist. Литература 1. Голубев Ю Л ., Вейн А М Неврологические синдромы Руководство для врачей Издательство Эйдос - Медиа . -2002.-832 с 2 Грачев Ю В ., Шмырев В И Тригеминальная лицевая боль : систематика клинических форм , принципы диагностики и лечения 10 с 3. Гречко В Е Неотложная помощь в нейростоматологии Москва , Медицина .-1981.- 200 с 4. Крыжановский Г Н Центральные механизмы патологической боли // Тезисы Российской научно - практической конференции « Патологическая боль » 14-16 октября 1999 г . – Новосибирск , 1999 г .- с .1 5. Fricton JR. Critical commentary. A unified concept of idiopathic orofacial pain: clinical features. J Orofac Pain. 1999;13:185–189. 6. Merskey H, Bogduk N. Classification of Chronic Pain: Description of Chronic Pain Syndromes and Definitions of Pain Terms. Seattle, WA: IASP Press; 1994. 7. Headache classification committee of the international headache society. Classification and diagnostic criteria for headache disorders, cranial neuralgias, and facial pain. \\Cephalalgia. 1998;8:1–96. 8. Okeson JP, ed. Orofacial Pain: Guidelines for Assessment, Diagnosis and Management. Chicago, IL: Quintessence; 1996. 9. Bennett GJ. Neuropathic pain. In: Wall PD, Melzack RM, eds. Textbook of Pain. London, Edinburgh: Churchill Livingston; 1994:201–224. 10. Eide PK, Rabben T. Trigeminal neuropathic pain: pathophysiological mechanisms examined by quantitative assessment of abnormal pain and sensory perception.\\ Neurosurgery. 1998;43:1103–1109. 11. Ren K, Dubner R. Central nervous system plasticity and persistent pain.\\ J Orofac Pain. 1999;13:155–163. 12. Fromm GH, Sessel BJ. Trigeminal Neuralgia. Current Concepts Regarding Pathogenesis and Treatment. Boston, MA: Butterworth- Heinemann; 1991. 13. Massey EW, Massey J. Elongated styloid process (Eagles Syndrome) causing hemicrania. Headache. 1979;19:339 –341. 14. Janetta PJ. Observation on the etiology of trigeminal neuralgia, hemifacial spasm, acoustic nerve dysfunction and glossopharyngeal neuralgia. Definitive microsurgical treatment and results in 117 patients.\\ Neurochirurgia. 1977; 20:145–154. 15. Vickers RE, Cousins MJ, Walker S, et al. Analysis of 50 patients with atypical odontalgia\\ цитир по Graff-Radford The Neurologist • Volume 15, Number 4, July 2009. 16. Solberg WK, Graff-Radford SB. Orodental considerations of facial pain.\\Semin Neurol. 1988;8:318 –323. 17. Bennett GJ. Neuropathic pain. In: Wall PD, Melzack RM, eds. Textbook ofPain. London, Edinburgh: Churchill Livingston; 1994:201–224. 18. Bennett GJ, Xie YK. A peripheral mononeuropathy in rat that produces disorders of pain sensation like those seen in man. \\Pain. 1988;33:87–107. 19. Coyle DE, Sehlorst CS, Mascari C. Female rats are more susceptible to the development of neuropathic pain using the partial sciatic nerve ligation(PSNL) model.\\ Neurosci Lett. 1995;186:135–138. 20. Coyle DE, Selhorst CS, Behbehani MM. Intact female rats are more susceptive to the development of tactile allodynia than overiectomized female rats following partial sciatic nerve ligation (PSNL).\\ Neurosci Lett.1996;203:37– 40. 21. Roberts AM, Person P. Etiology and treatment of idiopathic trigeminal and atypical facial neuralgias.\\ Oral Surg. 1979;48:298 –307. 22. Symonds C. Facial Pain. \\Ann R Coll Surg Eng. 1949;4:206 – 212. 23. Fromm GH, Graff-Radford SB, Terrence CF. Pre-trigeminal neuralgia.\\ Neurology.1990;40:1493–1495. 24. Woolf CJ, Mannion RJ. Neuropathic pain: aetiology, symptoms, mechanisms, and management.\\ Lancet. 1999;353:1959 – 1964. 25. Watkins LR. “Listening” and “Talking” to neurons: implications of immune activation for pain control and increasing efficacy of opioids.\\ Brain Res Rev.2007;56:48–69. 26. Janig W, McLAchlan EM. The role of modifications in noradrenergic peripheral pathways after nerve lesions in the generation of pain.\\ Progress in Pain Research and Management. Vol 1.Seattle, WA: IASP Press; 1994:101–128. 27. Campbell JN, Meyer RA, Davis KD, et al. Sympathetically mediated pain a unifying hypothesis. In: Willis WD, ed. Hyperalgesia and Allodynia, NewYork, NY: Raven Press; 1992:141– 149. 28. Roberts WJ, Foglesong ME. Identification of afferents contributing to sympathetically evoked activity in wide dynamic range neurons.\\ Pain. 1988;34: 305–314. 29. Scrivani SJ, Chaudry A, Maciewicz RJ, et al. Chronic neurogenic facial pain: lack of response to intravenous phentolamine. \\J Orofac Pain. 1999;13:89 –96. 30. Canavan D, Graff-Radford SB, Gratt BM. Traumatic dysesthesia of the trigeminal nerve.\\ J Orofac Pain. 1994;8:391–396. 31. Graff-Radford SB. Facial Pain of Undetermined Origin. In Rappaport A,Sheftell F, Purdy A, eds. Advanced Therapy of Headache: Case Based Strategies for Management. Hamilton, Ontario: BC Decker Publishers; 1999:263–269. 32. Woda A, Navez ML, Picard P, et al. A possible therapeutic solution for stomatodynia (burning mouth syndrome). \\J Orofac Pain. 1998;12:272–278. 33. Graff-Radford SB, Zarembinski C, Ananda AK, Hakimian B. Sphenopalatine ganglion block in traumatic trigeminal neuralgia and the outcome to Radiosurgical ablation. \\Medicine. 2009;10(1), 2. 34. Boas RA, Covino BG, Shahnarian A. Analgesic responses to IV lignocaine.\\Br J Anaesth. 1982;54:501–504. 35. Rowbotham MC, Reisner-Keller LA, Fields HL. Both intravenous lidocaine and morphine reduce the pain of postherpetic neuralgia\\Neurology. 1991;41: 1024–1028. 36. Sinnott CJ, Garfield JM, Strichartz GR. Differential efficacy of intravenous lidocaine in alleviating ipsilateral versus contralateral neuropathic pain in the rat. \\Pain. 1999;80:521–531. 37. Janetta PJ. Trigeminal neuralgia: treatment by microvascular decompression.In: Wilkins RH, Ragachary SS, eds. Neurosurgery. New York, NY: McGraw Hill; 1996:3961–3968. 38. Jarrahy R, Berci G, Shahinian HK. Endoscopic-assisted microvascular decompression of the trigeminal nerve. \\Otolaryngol Head Neck Surg. 2000;123:218 –223. 39. Young RF, Vermeulen Grimm P, et al. Gamma knife radiosurgery for treatment of trigeminal neuralgia. Idiopathic and tumor related.\\ Neurology.1997;48:608–614. 40. Edich RF, Winters KL, Britt L, et al. Trigeminal neuralgia. J Long Term Eff Med Implants. 2006;16:185–192. 41. Graff-Radford SB. SUNCT syndrome responsive to Gapbapentin (Neurontin).\\ Cephalalgia. 2000;20:515–517. 42. Schille H. Injuries of the temporomandibular joint: classification, diagnosis and fundamentals of treatment. In: Kruger E, Schilli W, eds. Oral and Maxillofacial Traumatology. Vol 2. Chicago, IL: Quintessence Publishing Co; 1986. 43. Takahashi T, Kondoh T, Fukuda M, et al. Proinflammatory cytokines detectable in synovial fluids from patients with temporomandibular disorders. Oral Surg Oral Med Oral Pathol \\Oral Radiol Endod. 1998;85:135–141. 44 Okeson JP. Diagnosis of temporomandibular disorders. In: Okeson JP, ed. Temporomandibular Disorder and Occlusion. 4th ed. St Louis, MO: Mosby; 1998:310 –351. 45. Isberg-Holm AM, Westesson PL. Movement of the disc and condyle in temporomandibular joints with clicking: an arthrographic and cineradiographic study on autopsy specimens. \\Acta Odontol Scand. 1982;40:151–164. 46. Farrar WB, McCarty WL. A clinical outline of the temporomandibular joint diagnosis and treatment. Montgomery, AL: NS Group; 1983:19. 47. Stegenga B, de Bont LG, Boering G, et al. Tissue responses to degenerative changes in the temporomandibular joint: a review.\\ J Oral Maxillofac Surg.1991;49:1079 –1088. 48. Westesson PL, Bifano JA, Tallents RH, et al. Increased horizontal angle of the mandibular condyle in abnormal temporomandibular joints. A magnetic resonance imaging study\\Oral Surg Oral Med Oral Pathol. 1991;72:359–363. 49 Nilner M, Petersson A. Clinical and radiological findings related to treatment outcome in patients with temporomandibular disorders. \\Dentomaxillofac Radiol. 1995;24:128 –131. 50. Nitzan DW. The process of lubrication impairment and its involvement in temporomandibular joint disc displacement: a theoretical concept.\\ J Oral Maxillofac Surg. 2001;59:36–45. 51. Scapino RP. The posterior attachment: its structure, function, and appearance in TMJ imaging studies. Part 1. J Craniomandib Disord. 1991;5:83–95. 52. Scapino RP. The posterior attachment: its structure, function, and appearance in TMJ imaging studies. Part 2.\\ J Craniomandib Disord. 1991;5:155– 66. 53. Dolwick MF. Intra-articular disc displacement. Part I: Its questionable role in temporomandibular joint pathology.\\ J Oral Maxillofac Surg. 1995;53:1069–1072. 54 de Bont LG, Dijkgraaf LC, Stegenga B. Epidemiology and natural progression of articular temporomandibular disorder.\\ Oral Surg Oral Med Oral Pathol. 1997;83:72–76. 55 Travell JG, Simons DG. Myofascial pain and dysfunction: the trigger point manual. Baltimore, MD: Williams and Wilkins Co; 1983. 56. Simons DG. Muscle pain syndromes, Part I.\\ Am J Phys Med. 1975;54:289–311. 57 Simons DG. Muscle Pain Syndromes, Part II\\Am J Phys Med. 1976;55:15– 42. 58. Travell JG. Myofascial trigger points: clinical view. In: Bonica JJ, Albe-Fessard D, eds. Advances in Pain Research and Therapy. New York, NY: Raven Press; 1976:919 –926. 59. Kellgren JH. Observations on referred pain arising from muscle.\\ Clin Sci. 1938;3:175–190. 60. Jaeger B. Myofascial referred pain patterns: the role of trigger points.\\ CDAJ. 1985;13:27–32. 61. Mense S. Referral of muscle pain new aspects. \\APS J. 1994;3:1–9. 62. Simons DG. Neurophysiological basis of pain, caused by trigger points. \\ APS J. 1994;3:17–19. 63. Fields HL, Heinricher M. Brainstem modulation of nociceptor-driven withdrawal reflexes. \\Ann NY Acad Sci. 1989;563:34–44. 64. Olesen J. Clinical and pathophysiological observations in migraine and tension type headache explained by integration of vascular, suprapinal and myofascial inputs.\\ Pain. 1991;46:125–132. 65. Ernberg M, Hadenberg-Magnusson B, Alstergren P, et al. Pain, allodynia, and serum serotonin level in orofacial pain of muscular origin.\\ J Orofac Pain.1999;13:56–62. 66. Shah JP, Danoff JV, Desai MJ, et al. Biochemicals associated with pain and inflammation are elevated in sites near to and remote from active myofascial trigger points.\\ Arch Phys Med Rehabil. 2008;89:16 –23. 67. Graff-Radford SB, Reeves JL, Jaeger B. Management of headache: the effectiveness of altering factors perpetuating myofascial pain. \\Headache. 1987;27:186 –190. 68. Davidoff RA. Trigger points and myofascial pain: toward understanding how they affect headaches. \\Cephalalgia. 1998;18:436–438. Учебные пособия по неврологии в библиотеке НГМУ 1. Гусев , Евгений Иванович Неврология и нейрохирургия в 2 т .: учебник с приложением на компакт диске / Е И Гусев , А Н Коновалов , В И Скворцова .- М .: ЭОТАР - Медиа , 2007- ISBN 978-5-9704-0577-2 2. Триумфов , Андрей Васильевич Топическая диагностика заболеваний нервной системы : краткое руководство / А В Триумфов . 16- е изд . – М .: МЕДпресс - информ , 2009.- 264 с .- ISBN 5-98322-282-2 Руководство 3. C коромец , Александр Анисимович Нервные болезни : учебное пособие для студ Мед вузов / А А Скоромец , А П Скоромец , Т А Скоромец .- 2- е изд ., перераб и доп .- М .: МЕД пресс - информ , 2007.-552 с .: ил . ISBN 5-98322-277-5 4. Нервные болезни : учебник для стом Фак мед вузов / М Н Пузин ( и др .); ред М Н Пузин .- М .: Медицина , 1997.- 336 с 5. Болезни нервной системы : руководство для врачей в 2 т / ред Н Н Яхно .- М .; Медицина ,2005.-4- е изд ., перераб и доп .- 512 с 6. Карлов Владимир Алексеевич Неврология лица / В А Карлов .- Мю : Медицина , 1991.- 286 с 7. Гречко Владислав Евдокимович . Неотложная помощь в нейростоматологии / В . Е . Гречко .- 2- е изд . Перераб . и доп .- М .: Медицина ,1990.-256 с . 8. Мументалер М Неврология : пер с нем ./ М Мументалер , Х Маттле ; Ред О С Левин .-2- е изд .- М .: МЕДпресс - информ , .- 2009.-920 с .: ид .- Руководство 9. Штульман Д Р Неврология : справочник практического врача / Д Р Штульман , О С Левин .- 6- е изд Доп и перераб .- М .: МЕДпресс - информ ,2008.-1024 с 10. Можаев С В Нейрохирургия ; учебник для вузов / С В Можаев , А А Скоромец , Т А Скоромец .-2- е изд ., перераб и доп .- М .: ГЭОТАР - Медиа ,2009.- 480 с .: ил УЧД - Учебник , УЧД - Рекомендовано медсоветом ВУЗа |